Definitions

1. Tachycardia is a fast regular rhythm; bradycardia is a slow rhythm.
2. Tachycardia is a resting heart rate more than 100 beats per minute.
3. Tachycardia is the medical term for an abnormally fast heart rate.
4. Tachycardia: A rapid heart rate, usually defined as greater than 100 beats per minute.
5. Tachycardia is an abnormally rapid beating of the heart, defined as a resting heart rate of over 100 beats per minute.

Tachycardias

1. In extreme cases, tachycardia can be life threatening.
2. This tachycardia can cause palpitations or, rarely, tachycardia-related cardiomyopathy ( 96).
3. Tachycardias that occur with sick sinus syndrome are characterized by a long pause after the tachycardia.
4. The tachycardias include sinus tachycardia, paroxysmal atrial tachycardia (PAT), and ventricular tachycardia.
5. Tachycardias are common cardiac problems in dogs, and atrial paroxysmal tachycardia is a serious cardiac arrhythmia that may lead to syncope.

Ventricular Tachycardia

1. Ventricular tachycardia is a potentially lethal arrhythmia and may result in an absent pulse.
2. The ICD may also be programmed to send a rapid burst of paced beats to interrupt the ventricular tachycardia.
3. Ventricular tachycardia (VT or V-tach) is a potentially life-threatening cardiac arrhythmia that originates in the ventricles.
4. Cardiac Disorders pulmonary edema, cardiac arrest or failure, tachycardia, ventricular fibrillation, and ventricular tachycardia.
5. Tachycardia can occur in either the upper heart chambers (atrial tachycardia) or lower heart chambers (ventricular tachycardia).

Rhythm

1. Sinus rhythm or sinus tachycardia with a normal mean electrical axis are typical.
2. The cardiac rhythm may be normal sinus, sinus tachycardia, or atrial fibrillation.
3. Class IIa Pharmacological therapy can be useful in patients with AF to maintain sinus rhythm and prevent tachycardia-induced cardiomyopathy.
4. In some people, tachycardia is the result of a cardiac arrhythmia (a heart-generated abnormality of heart rate or rhythm).
5. Examination of the heart revealed marked tachycardia and a regular rhythm, without gallops or murmur.

Accessory Pathway

1. An irregular, sustained, wide-QRS-complex tachycardia suggests AF with conduction over an accessory pathway or AF with bundle-branch block.
2. D. Atrioventricular reciprocating tachycardia (Extra nodal accessory pathways).

Av Node

1. Supraventricular tachycardia (SVT), which is a tachycardia paced from the Atria or the AV node.
2. The key to stopping the tachycardia is interrupting the rapid electrical impulse within the AV node.
3. Supraventricular tachycardia (SVT), which is a tachycardia paced from the atria or the AV node.
4. In supraventricular tachycardia, the heart rate is sped up by an abnormal electrical impulse starting in the atria.
5. As is typical in other reentry tachycardias, electrical cardioversion terminates this type of atrial tachycardia.

Catheter Ablation

1. Transesophageal echocardiography during radiofrequency catheter ablation of ventricular tachycardia.
2. Ten of 11 patients undergoing RF catheter ablation in this series had acute tachycardia elimination.
3. If you have tried other treatment, such as medicine and catheter ablation, but still have tachycardia, a pacemaker might be an option.
4. Catheter ablation can eliminate atrioventricular nodal reciprocating tachycardia (AVNRT), a type of supraventricular tachycardia, in almost all cases.
5. Catheter ablation is occasionally used in conjunction with ICDs in patients who experience multiple episodes of ventricular tachycardia.

Sinus Tachycardia

1. The treatment of inappropriate sinus tachycardia is predominantly symptom driven.
2. Sinus Tachycardia is not associated with an abnormal electrical pattern on the electrocardiogram.
3. B) sinus tachycardia with frequent couplets of atrial premature depolarizations after cardioversion (130 beats•min -1).
4. A harmless rhythm, sinus tachycardia is a normal increase in heart rate that happens with fever, excitement and exercise.
5. Causes of sinus tachycardia may include exercise, anxiety, fever, drugs, anemia, heart failure, hypovolemia and shock.

Heart Rate

1. An arrhythmia is any disorder of your heart rate or heart rhythm, such as beating too fast (tachycardia), too slow (bradycardia), or irregularly.
2. Sinus tachycardia is the term used to describe a resting heart rate of more than 100 beats per minute.
3. In SSS, the heart rate can switch back and forth between a slow rate (bradycardia) and a fast rate (tachycardia).
4. An abnormal increase in heart rate is referred to as tachycardia within the medical community.
5. Regular treatment of ventricular tachycardia includes medications to slow the heart rate.

Atrial Tachycardia

1. A third form of SVT, known as atrial tachycardia, arises from a circuit in the upper chambers (atria) of the heart.
2. Multifocal atrial tachycardia.
3. Atrial tachycardia, usually with AV block, may be produced by digitalis excess.
4. Atrial fibrillation is common atrial tachycardia, PSVT that-s the AV nodal reentrant rhythms.
5. Under such a circumstance, the tachycardia is very fast and may mimic atrial tachycardia or flutter.

Regular

1. It tends to be a regular, narrow complex tachycardia and may be a sign of digitalis toxicity.
2. It is usually a regular, wide complex tachycardia with a rate between 120 and 250 beats per minute.
3. It is a regular narrow complex tachycardia that usually responds well to vagal maneuvers or the drug adenosine.
4. Chaotic atrial activity produces a narrow-complex tachycardia with no regular P waves.
5. This results in a narrow-complex tachycardia with no regular P waves but a fibrillatory baseline and an irregular rhythm (see ECG below).

Sinus Node

1. If the tachycardia originates from the sinus node (sinus tachycardia), treatment of the underlying cause of sinus tachycardia is usually sufficient.
2. The most common type of tachycardia is sinus tachycardia, which is the body's normal reaction to stress, including fever, dehydration, or blood loss (shock).
3. There have been no controlled trials of drug prophylaxis involving patients with sinus node re-entrant tachycardia.
4. Sinus Tachycardia is due to rapid firing of a the sinoatrial (sinus) node, which is the natural pacemaker of the heart.
5. ICDs are used for fast arrhythmias such as ventricular tachycardia or ventricular fibrillation.

Ventricular Fibrillation

1. Defibrillation is a technique used in emergency medicine to terminate ventricular fibrillation or pulseless ventricular tachycardia.
2. While ventricular tachycardia can lead to ventricular fibrillation, it may break on its own, allowing the person's normal rhythm to resume.

Heart Beats

1. Abnormal slowing of the heart beat (called Bradycardia) and abnormal fast heartbeats, (called Tachycardia).
2. When the heart beats faster than normal, it is called tachycardia.
3. A normal heart beats 60 to 100 times a minute, but a heart with ventricular tachycardia may beat 160 to 240 times a minute.
4. If the heart beats more than 100 times per minute, the heart rate is considered fast (tachycardia).
5. When the heart beats too fast, it's called tachycardia.

Supraventricular Tachycardia

1. It is used in recurrent atrial fibrillation and other types of supraventricular tachycardia.
2. Heart rate is a predictor of success in the treatment of adults with symptomatic paroxysmal supraventricular tachycardia.
3. A supraventricular tachycardia (SVT) is a rapid rhythm of the heart in which the origin of the electrical signal is either the atria or the AV node.
4. Supraventricular tachycardia (SVT) is an abnormal fast heart rhythm that starts in the upper chambers, or the atria, of the heart.
5. Supraventricular tachycardia (SVT) is also called paroxysmal supraventricular tachycardia (PSVT) or paroxysmal atrial tachycardia (PAT).

Sustained Ventricular

1. Ventricular tachycardia may not cause symptoms in some people, but may be lethal in others -- it is a major cause of sudden cardiac death.
2. Ventricular tachycardia may be an emergency situation and require immediate CPR, electrical shock (defibrillation), or anti-arrhythmic medications.
3. An implantable cardioverter defibrillator is used in patients at risk for recurrent, sustained ventricular tachycardia or fibrillation.
4. New devices also provide overdrive pacing to electrically convert a sustained ventricular tachycardia, and "backup" pacing if bradycardia occurs.
5. ECG changes that have been detected after administration of anthracyclines include supraventricular dysrhythmias, heart block, and ventricular tachycardia.

Junctional

1. Junctional tachycardia is an automatic tachycardia originating in the AV junction.
2. Four patients needed no therapy because of the infrequency of permanent junctional re-entry tachycardia episodes.
3. The ECG features of focal junctional tachycardia include heart rates of 110 to 250 bpm and a narrow complex or typical BBB conduction pattern.
4. Procainamide for rate control of postsurgical junctional tachycardia.

Radiofrequency Ablation

1. Identification of reentry circuit sites during catheter mapping and radiofrequency ablation of ventricular tachycardia late after myocardial infarction.
2. Das MK, Miller JM. Pace mapping for radiofrequency ablation of ventricular tachycardia.
3. Unfavourable RF ablation outcomes were predefined as unsuccessful RF ablation or recurrence of tachycardia requiring repeated ablation.
4. Radiofrequency ablation has revolutionized the treatment of tachycardia caused by a re-entrant pathway.
5. Long-term follow-up in patients with the permanent form of junctional reciprocating tachycardia treated with radiofrequency ablation.

Tachycardia-Induced Cardiomyopathy

1. Patients with tachycardia-induced cardiomyopathy may be at long-term risk for sudden death.
2. Patients with an uncontrolled ventricular response during AF may occasionally develop a tachycardia-induced cardiomyopathy.

Termination

1. For patients with automatic AT, atrial pacing (or adenosine) may result in transient postpacing slowing but no tachycardia termination.
2. For termination of an irregular wide QRS-complex tachycardia (ie, pre-excited AF), DC cardioversion is recommended.

Paroxysmal Atrial

1. The tachycardias include sinus tachycardia, paroxysmal atrial tachycardia (PAT), and ventricular tachycardia.
2. Treatment of refractory paroxysmal atrial tachycardia during 3 successive pregnancies.

Rapid

1. However, ventricular tachycardia usually results in rapid heart rates that prevent the heart from pumping blood effectively.
2. Ventricular tachycardia can occur in episodes during which the person will have a rapid pulse or the symptoms described above.

Fast Heart

1. Ventricular tachycardia - a fast heart rate that originates in the lower chambers (ventricles).
2. Supraventricular tachycardia - a fast heart rate that originates in the upper chambers (atria).
3. Rhythm abnormalities can also be visualized as in slow heart rate bradycardia, or fast heart rate tachycardia.
4. This weakened condition, termed chronotropic cardiomyopathy, is usually a result of a long period of tachycardia(fast heart rate).

Atrioventricular Nodal

1. The most common are atrial fibrillation or flutter, and atrioventricular nodal reentry tachycardia.
2. B. Atrioventricular nodal reciprocating tachycardia.

Premature Ventricular

1. Ventricular tachycardia is a rapid heart beat initiated within the ventricles, characterized by 3 or more consecutive premature ventricular beats.
2. Ventricular tachycardia was detected from Holter monitoring in 19 patients.
3. During the episodes, monitor ECG showed two times of ventricular tachycardia and one of paroxysmal A-V block.
4. It has been reported to cause sinus bradycardia, heart block, premature ventricular contractions, and ventricular tachycardia.

Tachycardia Ventricular

1. All (with the possible exception of amiodarone) increase the risk of ventricular tachycardia, which can be fatal.
2. A common mechanism for ventricular tachycardia is reentry (re-stimulation of the electrical conductive pathway from a single initial stimulus).
3. ICDs are typically used for fast arrhythmias such as ventricular tachycardia or ventricular fibrillation.
4. An ICD is a device used primarily to treat ventricular tachycardia and ventricular fibrillation, two life-threatening heart rhythms.
5. Ventricular tachycardia (VT): A condition in which an electrical signal is sent from the ventricles at a very fast but often regular rate.

Tachycardia

1. Arrhythmia may be classified by rate (normal, tachycardia, bradycardia), or mechanism (automaticity, reentry, fibrillation).
2. Abnormal heartbeats occur when the heart has an irregular heart rhythm, beats too fast (tachycardia), or beats too slow (bradycardia).
3. An arrhythmia is a disorder of the heart rate (pulse) or heart rhythm, such as beating too fast (tachycardia), too slow (bradycardia), or irregularly.
4. Defibrillation is the definitive treatment for the life-threatening cardiac arrhythmias, ventricular fibrillation and pulseless ventricular tachycardia.
5. Patients with acute and severe anemia appear in distress with tachycardia, tachypnea, and hypovolemia.

Hypertension

1. These patients often have a hyperadrenergic response to posture, with both orthostatic tachycardia and hypertension.

Headache

1. Dihydropyridine CCBs can cause flushing, headache, excessive hypotension, edema and reflex tachycardia.
2. Adverse reactions to epinephrine include palpitations, tachycardia, anxiety, headache, tremor, hypertension, and acute pulmonary edema.
3. Neuroretinitis, chest pain, headache, allergic reaction, hypotension and tachycardia have been reported as part of ongoing surveillance.

Dizziness

1. Symptoms of ventricular tachycardia include light headedness, dizziness, fainting, shortness of breath and chest pains.
2. There was a feeling of intense heat, itching and burning of the skin, dizziness, tachycardia, chest pain, headache, and decreased vision.
3. Beta-blockers may mask tachycardia occurring with hypoglycemia, but other manifestations such as dizziness and sweating may not be significantly affected.

Angina

1. Tachycardia can cause various heart-related problems like myocardial infarction, angina and ischemic heart disease.
2. Depending on the severity of the anemia, fatigability, palpitations, dyspnea, angina, and tachycardia may occur.
3. Primary side effects include those commonly seen for β 1 active sympathomimetics, such as hypertension, angina, arrhythmia, and tachycardia.

Exercise

1. Underlying physiological stresses such as hypoxia, hypovolemia, fever, anxiety, pain, hyperthyroidism, and exercise usually induce sinus tachycardia.
2. During exercise or emotional stress, impaired calcium regulation in the heart can lead to ventricular tachycardia in people with CPVT.
3. Symptoms include weakness, reluctance to exercise, pale mucus membranes, and tachycardia (abnormally rapid heart rate).

Fever

1. Further symptoms include abdominal distention, nausea, vomiting, fever, tachycardia, diaphoresis, and jaundice.
2. Adverse events reported were dyspnea, malaise, nausea, fever, rash, sinus tachycardia, headache and chills.
3. A late sign in the acute phase of this syndrome, tachypnea typically accompanies anorexia, insomnia, tachycardia, fever, and diaphoresis.

Shock

1. Also, patients taking beta-blockers may not present with tachycardia, regardless of the degree of shock.
2. Physical examination reveals fever, hypotension, shock, tachycardia, and new murmurs or rubs (or recent changes in previously detected murmurs).
3. Signs of shock (such as pallor, tachycardia, and hypotension) may also appear.

Rapid Rate

1. Ventricular tachycardia is a rapid rate that originates with abnormal electrical signals in the ventricles.
2. This condition, called AV reentrant tachycardia, sends impulses to the ventricles at a very rapid rate.

Automaticity

1. Finally, Junctional Ectopic Tachycardia or JET is a rare tachycardia caused by increased automaticity of the AV node itself initiating frequent heart beats.
2. Atrial induction of ventricular tachycardia: reentry versus triggered automaticity.
3. Multifocal atrial tachycardia occurs when multiple sites in the atria are discharging and is due to increased automaticity.

Reentry

1. This process, called reentry, is a common cause of tachycardia.
2. Reentry as a cause of ventricular tachycardia in patients with chronic ischemic heart disease: electrophysiologic and anatomic correlation.
3. A common mechanism for ventricular tachycardia is reentry (re-stimulation of the electrical conductive pathway from a single initial stimulus).

Lower Chambers

1. Ventricular tachycardia - a fast heart rate that originates in the lower chambers (ventricles).
2. Ventricular tachycardia Ventricular tachycardia occurs when the ventricles (the lower chambers of the heart) beat too fast.
3. Ventricular tachycardia is a rapid, regular heart rhythm that originates in the lower chambers of the heart.

Medications

1. A fast heart rate (tachycardia) may be treated with medications.
2. In some cases the only way to avoid paroxysmal atrial tachycardia is through medications or through a surgery called cardiac ablation.
3. Other causes of ventricular tachycardia include heart disease and medications.

Cardioversion

1. Patients with a stable VT are given cardioversion if the tachycardia exceeds 150bpm.
2. Cardioversion For tachycardia with serious signs and symptoms.
3. Ventricular tachycardia should be handled by cardioversion and, if persistent, by overdrive pacing.

Treatment

1. Digoxin is is not indicated for the treatment of sinus tachycardia unless it is associated with heart failure.
2. If you have very frequent PVCs associated with underlying heart disease and periods of ventricular tachycardia, your doctor might recommend other treatment.
3. The treatment of paroxysmal supraventricular tachycardia in such patients is usually direct-current cardioversion.

Verapamil

1. Adenosine for paroxysmal supraventricular tachycardia: dose ranging and comparison with verapamil.
2. Response to verapamil is an important feature of fascicular tachycardia.
3. Effects of verapamil and lidocaine on two components of the re-entry circuit of verapamil-sensitive idiopathic left ventricular tachycardia.

Wpw

1. Other people with WPW never have tachycardia or other symptoms.
2. WPW is a type of atrioventricular reentrant tachycardia.

Ecg

1. If an electrocardiogram (EKG, ECG) can be performed while ventricular tachycardia is occurring, it often provides the most useful information.
2. An ECG done during a spell of tachycardia almost always shows the abnormal rhythm.
3. If your tachycardia seems related to exercise, you may need to have an ECG while exercising on a treadmill.

Qrs Complex

1. They selected the area where a Purkinje potential precedes the QRS complex during tachycardia.
2. P waves always follow the QRS complex in orthodromic reciprocating tachycardia of WPW syndrome.
3. The QRS complex in ventricular tachycardia often has a right or left bundle branch morphology.

Underlying Mechanism

1. We conclude that delayed termination of tachycardia is not an indicator of the underlying mechanism of tachycardia.
2. The underlying mechanism may be vasodilatation either directly or indirectly owing to release of histamine with reflex tachycardia.
3. Digitalis intoxication is one of the important causes of atrial tachycardia, with triggered activity as the underlying mechanism.

Heart Rates

1. Heart rates of less than fifty beats per minute are defined as bradycardia and rates above one hundred are defined as tachycardia.
2. The ECG features of focal junctional tachycardia include heart rates of 110 to 250 bpm and a narrow complex or typical BBB conduction pattern.

Categories

1. Society > Humans > Medicine > Heart Rate
2. Health > Diseases > Symptoms > Bradycardia
3. Health > Diseases > Heart Disease > Arrhythmia
4. Fast
5. Glossaries > Glossary of Cardiology /

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