Definitions

1. H pylori gastritis is the most frequent cause of MALT lymphoma. (Web site)
2. H pylori gastritis is usually acquired during childhood, and complications typically develop later. (Web site)
3. H. pylori gastritis is caused by infection from the H. pylori bacterium. (Web site)
4. H. pylori gastritis is easily diagnosed through the use of the urea breath test. (Web site)
5. Acute H. pylori gastritis is superficial pangastritis progressing into the chronic phase after 7-10 days.

Pylori Dna

1. METHODS: H. pylori DNA was extracted from antral biopsy specimens from 33 children with H. pylori gastritis. (Web site)

Human Studies

1. Seven of 9 human studies showed an improvement of H. pylori gastritis and decrease in H. pylori density after administration of probiotics. (Web site)

Gastric Bypass

1. Occasionally, individuals with H. pylori gastritis, total or partial gastrectomy, or gastric bypass may develop a B 12 deficiency. (Web site)

Triple Therapy

1. H pylori gastritis usually responds well to triple therapy of amoxycillin, metronidazole and deNol. (Web site)

Humans

1. If there were many intriguing similarities between H. pylori gastritis in gerbils and humans, there were also some notable differences.
2. An animal model of Helicobacter pylori gastritis in humans.

Antibiotics

1. The eradication of H. pylori gastritis by antibiotics has been shown to cure peptic ulcers and prevent recurrence (4,5). (Web site)
2. Inducible nitric oxide synthase, nitrotyrosine, and apoptosis in Helicobacter pylori gastritis: effect of antibiotics and antioxidants.

Pathogenesis

1. The role of T cell subsets and cytokines in the pathogenesis of Helicobacter pylori gastritis in mice.

Results

1. Long-term course and consequences of Helicobacter pylori gastritis: results of a32-year follow-up study.

Peptic Ulcer Disease

1. With prolonged antibiotic therapy, H. pylori gastritis and peptic ulcer disease can often be cured.

Gastric

1. Profound suppression of gastric acid is associated with increased severity of H pylori gastritis.
2. CONCLUSION: Among men between 55 and 65 years of age, the gastric cancer phenotype of H. pylori gastritis appears to be more common than expected.
3. The gastric H+,K+-ATPase is a major autoantigen in chronic Helicobacter pylori gastritis with body mucosa atrophy. (Web site)

Patients

1. TRIPLE THERAPY. As of early 1998, triple therapy was the preferred treatment for patients with H. pylori gastritis. (Web site)
2. Differing patterns of Helicobacter pylori gastritis in patients with duodenal, prepyloric, and gastric ulcer disease. (Web site)

Antrum

1. The results of this study show a different expression of H. pylori gastritis in the antrum and body, which is the main subtype of chronic type B gastritis.
2. Apoptosis in different compartments of antrum and corpus mucosa in chronic Helicobacter pylori gastritis. (Web site)

Helicobacter Pylori Gastritis

1. Differences in distribution and severity of Helicobacter pylori gastritis in children and adults with duodenal ulcer disease. (Web site)
2. Helicobacter pylori gastritis can also progress to atrophic gastritis of the antrum, which increases the risk of peptic ulceration and stomach cancer.
3. Helicobacter pylori gastritis in children is associated with a regulatory T-cell response.

Pylori Gastritis

1. The pathogenesis of peptic ulcer and gastric cancer is closely associated with H. pylori gastritis and its subsequent atrophic sequelae (atrophic gastritis).
2. In addition, nearly all patients with duodenal ulcer disease have H. pylori gastritis, and ulcer relapse is exceptional after H. pylori eradication.
3. Patients with H pylori gastritis are 3-6 times more likely to develop gastric cancer than individuals without the infection. (Web site)

Categories

1. Helicobacter Pylori Gastritis
2. Duodenal Ulcer Disease
3. Antrum
4. Triple Therapy
5. Encyclopedia of Keywords > Society > Humans > Atrophic Gastritis
6. Books about "Pylori Gastritis" in Amazon.com
   

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