Definitions

1. Erythropoiesis is the process by which red blood cells (erythrocytes) are produced. (Web site)
2. Erythropoiesis is the development process in which new erythrocytes are produced, through which each cell matures in about 7 days. (Web site)
3. Erythropoiesis, the production of RBCs, starts in the red bone marrow with a precursor cell called a proerythroblast.
4. Erythropoiesis is controlled by red blood cell growth factors.
5. The rate of erythropoiesis is measured by a reticulocyte count.

Associated

1. The absence of dietary histidine is associated with failure of normal erythropoiesis. (Web site)

Macrophages

1. Late in erythropoiesis, nuclei are expelled from the erythroid precursor cells and engulfed by the macrophages in the blood island.

Inhibition

1. Hepcidin mRNA levels in mouse liver respond to inhibition of erythropoiesis.

Subsequent Development

1. Chronic exposure to arsenic has been associated with disrupted erythropoiesis with subsequent development of anemia, leucopenia, and thrombocytopenia.

Iron Status

1. Beguin Y. Soluble transferrin receptor for the evaluation of erythropoiesis and iron status. (Web site)

Ferroportin

1. Our results showed a coordinated upregulation of ferroportin in enterocytes and macrophages in response to iron-restricted erythropoiesis. (Web site)

Heme Iron

1. Daily iron required for erythropoiesis is predominantly derived from recycling of heme iron by macrophages erythrophagocytosing senescent RBCs.

Regulation

1. Disruption of ferroportin 1 regulation causes dynamic alterations in iron homeostasis and erythropoiesis in polycythaemia mice.

Red Blood Cell Production

1. Figure 2. Regulation of red blood cell production (erythropoiesis). (Web site)

Myelodysplastic Syndromes

1. T-cell mediated inhibition of erythropoiesis in myelodysplastic syndromes. (Web site)
2. T cell-mediated inhibition of erythropoiesis in myelodysplastic syndromes.

Iron Absorption

1. Delayed hepcidin response explains the lag period in iron absorption following a stimulus to increase erythropoiesis. (Web site)
2. There is a direct correlation between the level of erythropoiesis and iron absorption.

Diamond-Blackfan Anemia

1. Diamond-Blackfan anemia is a congenital disorder of erythropoiesis in humans, characterized by a macrocytic anemia often associated with physical anomalies. (Web site)
2. Response of Diamond-Blackfan anemia to metoclopramide: evidence for a role for prolactin in erythropoiesis. (Web site)

Bone Marrow

1. In humans, erythropoiesis occurs almost exclusively in the bone marrow. (Web site)

Effects

1. Nyagolov Y, Dyankov E and Ganchev T. Effects of piracetam on erythropoiesis and leukopoiesis in rats.

Effect

1. Effect of iron stores and erythropoiesis. (Web site)
2. Effect of parathyroid hormone on erythropoiesis. (Web site)
3. Possible mechanisms underlying the effect of natural preparations on erythropoiesis under conditions of conflict situation.: Bull Exp Biol Med. (Web site)

Production

1. This process normally occurs at the same rate of production by erythropoiesis, balancing the total circulating red blood cell count. (Web site)

Ribosomes

1. Erythrocytes mature through erythropoiesis in the bone marrow, where they lose their nuclei, organelles, and ribosomes. (Web site)

Iron

1. The intestinal iron absorption is modulated in response to the level of body iron stores and by the amount of iron needed for erythropoiesis (11). (Web site)
2. Transferrin-bound iron is utilized mainly for erythropoiesis at the bone marrow, in which iron is essential for the formation of heme. (Web site)

Rbc

1. The primary reason to assess the RBC is to check for anemia and to evaluate normal erythropoiesis (the production of red blood cells).

Rbcs

1. This type of anemia is caused by deficient erythropoiesis, the ongoing process of the bone marrow to produce healthy red blood cells (RBCs). (Web site)

Hepcidin

1. Hepcidin contributes to AI by shunting iron away from erythropoiesis and sequestering it in the liver, predominantly in hepatocytes. (Web site)

Hypoxia

1. It is known that hepcidin is regulated by iron, hypoxia, inflammation, and erythropoiesis.

Erythropoietin

1. RBC production or, erythropoiesis, is stimulated when certain kidney cells detect low blood oxygen levels and release a hormone (erythropoietin). (Web site)
2. Regulatory substances necessary for normal erythropoiesis include erythropoietin, thyroid hormones, and androgens. (Web site)

Anemia

1. Iron deficiency anemia occurs when iron deficiency is sufficiently severe to diminish erythropoiesis and cause the development of anemia. (Web site)
2. Potential role of parathyroid hormone as an inhibitor of erythropoiesis in the anemia of renal failure.
3. Negative regulation of hepcidin transcription Hypoxia, anemia, increased erythropoiesis and reduced iron stores all negatively regulate hepcidin expression. (Web site)

Red Blood Cells

1. It stimulates erythropoiesis (production of red blood cells) in the bone marrow. (Web site)
2. Cobalt, present in the body only in the form of vitamin B-12, is essential to erythropoiesis, or production of red blood cells. (Web site)
3. Red blood cells are produced in the bone marrow through a process called erythropoiesis. (Web site)

Erythrocytes

1. Erythrocytes are formed through the process known as erythropoiesis and leukocytes are formed through the process known as leukopoiesis.
2. Erythropoiesis: The production of erythrocytes.

Erythropoiesis

1. In an earlier chapter, we discussed how the red bone marrow participates in the formation of red blood cells (RBCs) through the process of erythropoiesis.
2. Hypoxia and anemia generally increase the production of erythropoietin, which in turn stimulates erythropoiesis. (Web site)
3. In anemia and hypoxia, hepcidin regulates the availability of iron for erythropoiesis.

Categories

1. Red Blood Cells
2. Erythrocytes
3. Red Bone Marrow
4. Anatomy > Tissues > Blood > Anemia
5. Iron Absorption
6. Books about "Erythropoiesis" in Amazon.com
   

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